Description
This track displays smoothed (sliding-window mean) scores for
regions bound by Sp1 and Sp3 in the following
three cell lines, assayed by ChIP and microarray hybridization:
Cell Line | Classification | Isolated From |
HCT 116 | colorectal carcinoma | colon |
Jurkat, Clone E6-1 | acute T cell leukemia | T lymphocyte |
K-562 | chronic myelogenous leukemia (CML) | bone marrow |
Display Conventions and Configuration
This annotation follows the display conventions for composite
tracks. The subtracks within this annotation
may be configured in a variety of ways to highlight different aspects of the
displayed data. The graphical configuration options are shown at the top of
the track description page, followed by a list of subtracks. To display only
selected subtracks, uncheck the boxes next to the tracks you wish to hide.
For more information about the graphical configuration options, click the
Graph
configuration help link.
Methods
Chromatin IP was performed as described in Trinklein et
al. (2004). Amplified and labeled ChIP DNA was hybridized to
oligo tiling arrays produced by NimbleGen along with a total genomic
reference sample. The data for each array were median subtracted (log
2 ratios) and normalized (divided by the standard deviation).
The transformed mean ratios of ChIP DNA:Total DNA for
all probes were then smoothed by calculating a sliding-window mean.
Windows of six neighboring probes (sliding two probes at a time) were
used; within each window, the highest and lowest value were dropped,
and the remaining 4 values were averaged. To increase the
contrast between high and low values for visual display, the average
was converted to a score by the formula:
score = 8^(average) * 10 .
These scores are for visualization purposes; for all analyses,
the raw ratios, which are available in the Stanf ChIP track, should be used.
Verification
Three biological replicates and two technical replicates were
performed. The Myers lab is currently testing the specificity and
sensitivity using real-time PCR.
Credits
These data were generated in the Richard M. Myers lab
at Stanford University (now at
HudsonAlpha Institute for Biotechnology).
References
Trinklein, N.D., Chen, W.C., Kingston, R.E. and Myers, R.M.
The role of heat shock transcription factor 1 in the
genome-wide regulation of the mammalian heat shock response.
Mol. Biol. Cell 15(3), 1254-61 (2004).
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